What is the primary reason for the rapid transport of a patient who ingested a large dose of Tylenol?

The primary reason for the rapid transport of a patient who ingested a large dose of Tylenol is that the administration of an antidote can prevent liver failure if it is given within a critical time frame. In cases of Tylenol overdose, the liver is at risk of significant damage due to the toxic metabolite N-acetyl-p-benzoquinone imine (NAPQI), which can deplete glutathione reserves and lead to hepatotoxicity. The antidote, N-acetylcysteine (NAC), works by replenishing glutathione stores and neutralizing NAPQI if administered promptly, ideally within 8 to 10 hours after ingestion. Therefore, immediate medical intervention is crucial to maximize the effectiveness of the antidote and minimize the risk of acute liver failure.

Options that discuss activated charcoal and cardiopulmonary arrest do not relate directly to the primary urgency associated with antidote administration, while the timing of liver failure occurring is important, it is the potential for prevention through the timely use of an antidote that is most significant for rapid transport.